http://www.stuff.co.nz/stuff/0,2106,2116651a7144,00.html
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Study shows some cot deaths are heart-related 20 November 2002 
        
CHICAGO: A study suggests 5 per cent of sudden infant death syndrome
cases may be due to an electrical problem in which the heart recharges
itself too slowly, researchers said today. 
About 3000 infants die each year in the United States alone of SIDS,
defined as the sudden and unexplained death of an infant less than 1 year
old. Previous research has also suggested sleeping on the stomach,
nervous system problems related to breathing, abnormal metabolism in the
liver and flaws in the heart's electrical channels could be among the
causes. 
The American Academy of Paediatrics recommends infants be placed on their
backs to sleep instead of on their stomachs, and studies have indicated
that change has reduced the incidence of SIDS. 
The study released today at the American Heart Association meeting in
Chicago found that in one out of 20 cases of SIDS, the baby has a problem
similar to a heart condition called long Q-T syndrome that sometimes
causes sudden death in young people and adults. 
With long Q-T syndrome, the heart electrically recharges itself too
slowly or in a disorganised way, ahead of the next heartbeat, and can
sometimes cause the heart to stop pumping. An estimated one in 5000
people may suffer from the syndrome. 
The researchers, led by Michael Ackerman, an assistant professor at the
Mayo Clinic, performed a genetic autopsy on every unexplained infant
death investigated in Arkansas from September 1997 to August 1999. There
were 93 cases. 
"Long Q-T syndrome is sometimes called the perfect killer, because it
leaves no clues," Ackerman said in a news release issued by the heart
association. "Neither does SIDS." 
"SIDS will probably turn out to have 20 different underlying causes," he
added. "If we can figure out what they are, we can screen for them and
hopefully one day, prevent future cases." 
Ackerman urged medical examiners to collect and store tissue in sudden
infant deaths in a way that allows researchers to carry out a molecular
autopsy, in order to continue to study the possible causes of SIDS. 

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