http://www.kuro5hin.org/story/2003/4/4/85519/43140

The end of malaria in sight? (Science)

By twistedfirestarter 
Fri Apr 4th, 2003 at 01:28:49 PM EST 
  
A biologist at Imperial College in London, Austin Burt, has suggested
(technical overview) a dramatic scheme for eradicating the world's most
deadly disease, malaria. He proposes the use of cutting-edge genetic
technology to make entire species of malaria-carrying mosquitoes of
extinct. This was widely reported in popular media around the time of
publication (20 March).

If his plan were to be realised, it could be the greatest application of
genetic science so far. It may also be akin to opening a Pandora's box,
giving humans the god-like power to annihilate entire species at will.
Either way, it is an important issue worthy of discussion, which is a
life and death issue for the millions who suffer from malaria each year.

Malaria background
At a time when many parts of the world are disproportionately fearful of
emergent diseases and biological or chemical attack, malaria is a mundane
but devastating killer that puts other causes of death in perspective.
The lack of interest in malaria may partly be because it is endemic in
Africa and India, some of the poorest places in the world. Every year,
1.5 - 2.7 million people (mostly children) die from malaria. It affects
tropical countries across the globe, and has plagued humanity for all
recorded history. It is also believed to have major economic consequences
for African nations in particular. 

Malaria mosquitos - Anopheles

An obvious, yet important, point is that not all mosquitos are malaria
mosquitos. The malaria mosquitos are classified as members of the genus
Anopheles. Anopheles is distributed throughout the world. It is the
subject of much interest in the genetic community. The genes for one
African species, A. gambiae, has recently been sequenced.

Mosquitos are any members of the family Culicidae. To wipe out Anopheles
would still leave other Culicidae species to fill any empty ecological
niches. This is important, as fish, frogs, turtles, birds and bats eat
mosquitoes. Mosquitos are also pollinators (many live off nectar rather
than blood). There may also be unknown roles played by mosquitos in the
ecology.

Homing Endonuclease Genes

HEGs are special genetic sequences found in the DNA of many species. They
are one of a number of [PDF] selfish genetic elements which exploit
various genetic processes to replicate themselves. HEGs exploit cells'
damage repair system to perpetuate themselves in the DNA.

A HEG, like many genes, codes for the production of an enzyme.  In this
case the enzyme is a homing endonuclease. Homing endonucleases catalyse
the movement of their own DNA sequence, which is the HEG, inside a
chromosome. 

A homing endonuclease will search for a particular sequence of DNA. What
DNA sequence it searches for is specified by the HEG. Once the homing
endonuclease enzyme has found this sequence, it will slice through the
DNA at that point, severing the chromosome in two. 

This leaves a "hole" in the genetic code that must be filled. In a
diploid cell with paired chromosomes, this problem can be fixed. The
nucleus will attempt to repair the chromosome by copying the
corresponding gene at the same point on the other chromosome in the pair
- clearly a sensible solution to the problem. But this is playing
straight into the HEG's hand - the HEG is this gene and is used as the
template to repair the break. 

Using HEGs to kill

HEGs, like most selfish genetic elements, are not harmful to the host.
Their survival relies upon the survival and reproductive success of their
host. This is because, unlike viruses, HEGs can only be transferred by
being passed down from one generation to the next - they are not
otherwise contagious. They never leave the DNA or produce carriers such
as viruses.  If the host dies without reproducing, the HEG will die with
it.

However, this does not mean that HEGs cannot be modified to be harmful to
their hosts by genetic engineering. The plan is to make the HEG target a
gene crucial for development in mosquitos, so that the enzyme will
destroy this gene when the HEG is activated. The HEG will only activate
within newly divided eggs or sperm cells, so that both kinds of cells
have 2 copies of the HEG and no copy of the normal gene. Because the HEG
copies itself in the sperm and egg cells, even though a carrier may only
have 1 copy of the HEG, their children must have at least 1 copy. This is
in apparent defiance of Mendelian genetics, and is why HEGs are different
from recessive traits.

When the HEG carriers reproduce with a non-HEG carrier, their children
will develop normally, but carry the HEG on one of the chromosomes. They
will live because they can use the gene on the other chromosome.  But if
a HEG carrier reproduces with another HEG carrier, the children will die,
as they will not have a copy of the crucial gene, just two copies of the
HEG. This delayed effect will mean that the HEG will be able to spread
throughout the mosquito population without the carriers being negatively
affected. This will be the case until carriers become predominant and
start mating with each other. At such time, however, if enough carriers
are in the population, a population crash could result as non-carrier
reproduction becomes rarer and rarer.

Burt's article predicts that after 12 generations, 80 per cent of a
population's offspring may be killed. This is if 1 per cent of the
mosquito population are given the HEG and it copies itself to the sperm
and egg cells in 95 per cent of cases. If the HEG can replicate in 99.9
per cent of cases or if more than one HEG is used, after 12 generations
99.8 per cent of a population's offspring will be killed. A mosquito
generation may be as short as 3 weeks in tropical areas, which means that
it may take only 36 weeks for near-extinction.

Resistance

There would be a simple way for mosquitos to evolve resistance. Because
the HEG will only home in on a specific DNA sequence, the mosquitos could
simply evolve a gene that has a different, non-matching, sequence but
performs essentially the same task.  This could be avoided by using a
number of HEGs, each targeting a different part of the same gene or
targeting a number of different, but equally crucial, genes. 

Resistance would only be an issue if the mosquitos were given enough time
to develop it. A large enough release of HEG carriers would lead to such
a swift population fall, this would hardly be an issue. It is worth
pointing out that using HEG to control a population is fundamentally
different from using a pesticide - it would not require repeated
exposures over many years, just a single release of a batch of carriers.
Given a large enough release, the chances of resistance forming can be
minimised by making sure the population crash is swift.

The same development which could allow mosquitos to be resistant to HEG
could be used to reverse the process once it has started. It could also
be used protect other species in case the HEG somehow jumped species. By
introducing a replacement gene, which is "immune" to the HEG, it would be
possible to reverse any damage which had been done. The carriers of the
new gene would have such a large reproductive advantage that the new gene
should spread quickly throughout the population.

Drawbacks

This proposal raises some difficult questions.  Why would we want to stop
with the malaria mosquito and not use the same techniques to remove other
deadly pests? There are many other diseases carried by insects (Tsetse
fly, West Nile virus). As we remove species after species would it may
become a more acceptable option, until removing mere annoyances such as
normal mosquitos or ticks would be a possibility.

Once we start wiping species out can we stop powerful interests groups
from using the same technology and rationale to make extinct other
species? Crop pests, for example, cause millions of dollars of damage (in
lost crops and cost of control) every year. Many farming interests would
see the HEG technique as a possible way to save money. There may be
environmental benefits from avoiding the use of pesticides, but the risk
of making species extinct should not be taken lightly. When there are
such powerful economic interests in favour of such action, would it be
possible to stop them? 

Other solutions

The HEG is just one possible technique of many for controlling malarial
mosquito populations. It should be considerably more effective and
powerful than other techniques, such as the release of sterile males. It
is also unproven, untested and entirely speculative. Additionally, the
release of genetically modified animals is hardly a popular technique.
However, the problem of malaria is so prevalent and widespread across the
globe that the most powerful techniques in the scientific arsenal may
need to be used.

There are more mundane solutions to the malaria problem: bed-netting can
save lives. Removing and banning stagnant water in which mosquitos breed
is another important public health measure. It is easy to forget the
simple but effective techniques when technological "silver bullets"
present themselves.

Conclusion

Malaria is a problem that we seem to have been inured to or have
forgotten about. It doesn't have the novelty of murder, war or disaster
even though it kills many millions of people every year. Yet it is an
important and significant issue, both in humanitarian and economic terms.
If malaria were to be wiped out, it would be a milestone in medicine and
genetic technology.

The answer to this entire question may seem obvious to many: Of course we
should use HEGs to eradicate malaria. But caution must be the rule when
making such monumental decisions as the future of an entire species. At a
time when many people are desperately trying to save species, to
contemplate the destruction of even the most harmful species seems
somewhat contradictory. 

In any case, it would be the first ever animal species destroyed by
people who had full knowledge of the possible consequences of such an
action. Humans have made hundreds species on the planet extinct but in
every case it was accidental, not a planned eradication. If we have it
within our power to make species extinct, we bear an awesome
responsibility.  We must be sure not to misuse it for economic, rather
than humanitarian, advantage.

References

New Scientist, 22 March 2003: Splat!, Oliver Morton
 


_______________________________________________
http://www.mccmedia.com/mailman/listinfo/brin-l

Reply via email to