Hi Zhijie,

Thank you so much for the reply. The protein I am working on is from eukaryotic 
cytosol. We've already tried optimize the IPTG concentration and induction 
temp. Co-expression of chaperons seems to be a interesting idea, I googled and 
found this one: 
http://www.clontech.com/takara/US/Products/Protein_Research/Protein_Folding_and_Expression/Chaperone_Plasmid_Set?sitex=10031:22372:US

Do u think it is good? Or do u have other recommendations?

Thank you very much!

Best,
Xiao



On 2013-11-14, at 下午5:14, Zhijie Li wrote:

> Hi Xiao,
>  
> The tail sequence does not look very hydrophobic. But until your experiments 
> is done we can’t really say for sure that is not the cause for the 
> aggregation.
>  
> If your protein has cys residues then if the protein molecules were misfolded 
> and were forming intermolecular disulfide links, adding 1-10mM DTT without 
> denaturation may not be able to resolve the disulfide linked multimers, 
> because the disulfide could be buried in the misfolded proteins. You can run 
> SDS PAGE with samples boiled with or without DTT to see if you have 
> intermolecular disulfide links.
>  
> Since your protein contains cys residues, I wonder what is the source of it. 
> Is it eukaryotic secreted protein or ER/Golgi protein? If the protein is 
> eukarytic cytosolic/nuclear protein you do not need to worry about the 
> oxidative folding of them, and producing in the cytosol of E. coli might be 
> fine. But if the protein was eukaryotic secreted protein that contain 
> disulfide bonds, then you have to find an expression system that have the 
> proper chaperone systems to let the peptide form correct disulfide linkages. 
> In such case, yeast, insect, or even mammalian expression system should be 
> tried.
>  
> The linker between your protein and MBP is important. You do need long enough 
> linker for making sure the two domains are not interfering each other’s 
> folding. This requirement is protein-specific though. The poly Asn linker 
> provided by the pMal vectors is normally long enough. According to your 
> description that your protein family members were not even soluble with other 
> tags, I think it is quite clear that these proteins are not well behaving in 
> your current expression system. So unless you detect serious problems in your 
> construct design, I do not recommend you to invest too much effort on trouble 
> shooting the linker and so on because this is likely not the main problem 
> here.
>  
> If you have to make it in e.coli, expression condition has to be optimized to 
> maximize their chance to fold into soluble proteins. These optimization may 
> include: 1) slow down the expression by using less IPTG or using lower temp; 
> 2) to co-expression chaperones(there are many reported cases this works like 
> magic); 3) to co-express the target protein’s binding partners/cofactors; 4) 
> to use special expression strains such as the NEB shuffle strain that allows 
> cytosolic oxidative folding of disulfide-containing proteins, or the rare 
> codon-expressing strains to resolve rare codon problems if any; 5) to send 
> your protein to periplasmic space (use pMAL-p vectors instead of pMAL-c) if 
> you believe the protein should contain disulfides
>  
> Or you can try to refold the protein in vitro. In such case you’d better use 
> the non-MBP tags, and get the inclusion bodies as the start point.
>  
> But after all, I always prefer to use expression systems that’s most close to 
> the protein’s natural habitat. There are proteins that would never be folded 
> in E coli.
> 
> Zhijie
>  
>  
>  
> From: Xiao Xiao
> Sent: Thursday, November 14, 2013 2:18 AM
> To: [email protected]
> Subject: Re: [ccp4bb] Oligomerization of maltose-binding protein
>  
> Thank you for the replies!
>  
> Rana: Thank you for your prompt reply! I have the same problem, based on my 
> SDS-gel, I also have that 42kD MBP band when purifying my fusion protein. 
> Fortunately I can separate that from my protein of interest by size-exclusion 
> column. So the major issue I have now is the oligomerization of my fusion 
> protein. And it is also weird that I got the free-MBP as a large oligomer.
>  
> Zhijie: Thank you for your very detailed and well-written suggestions. It is 
> very interesting that the MBP fusion protein can form 'micelles'. We found 
> very similar properties when cloning other family members into MBP vector, 
> they all have a very large oligomer peak, around 10-mer (several hundreds 
> kD), but not in void volumn of sup6 column. Biologically these proteins could 
> form oligomer but not this big. We have tried to purify these protein in 
> other tags, but most of them were insoluble. Only MBP tag gives us such an 
> impressive solubility. But I agree -- they may be still misfolded.
>  
> As I replied to Rana, I agree with you that the oligomerized free-MBP is 
> really strange. That's why I want to figure out the reason, to exclude the 
> possibility that the tag itself causes aggregation. Reading-frame should be 
> correct since SDS-gel indicates the peptide length is correct. I didn't put 
> the stop codon right after precision site, so there are some restriction 
> cutting sites got translated. The exact C-term should be 27aa 
> (LEVLFQGP+HMSMGGRDIVDGSEFPAGN). Do you think this could be the reason that 
> MBP get oligomerized? I am trying to delete these C-term tail to see how it 
> will behave.   
>  
> And the answer for your last question: there is one Cys supposed to be 
> reduced on the surface of my protein (others are internal and really 
> conserved), but I include 1mM DTT during purification and have tried even 
> 10mM DTT when running gel-filtration but it didn't get improved.
>  
> I have one more question. Let's say if the aggregation is really caused by 
> the property of this construct, could it be possible to help its folding by 
> modifying linker region between MBP tag and my protein? I know it could be 
> very tough, but is there any general idea or strategy that we can try as a 
> start point, such as change hydrophobicity?
>  
> Again, thank you all and I am looking for your responds!
>  
> Best regards,
> Xiao
> 
> 2013/11/13 Zhijie Li <[email protected]>
> Hi Xiao,
> 
> MBP usually is monomeric, unless you put something really nasty to its ends. 
> People have mentioned before that due to the high solubility of MBP, MBP tag 
> can drag otherwise insoluble/overly hydrophobic protein domains into 
> solution. Then this half hydrophilic half hydrophobic molecule can form 
> micelle-like structures.
> 
> 
> "the small peaks are both free-MBP monomer, but the big peaks are fusion 
> protein and free-MBP respectively."
> 
> So your fusion protein is never found in the monomer peak? That strongly 
> hints aggregation caused by the domain you fused to MBP. The free MBP monomer 
> peak you see on the MBP-fusion run is likely a proteolytic product of the 
> fusion protein, or is the natural MBP from E. coli (although normally far 
> less than the over expressed MBP fusion in quantity). Are you certain that 
> your target protein domain is not naturally oligomerizing? If it is not or 
> only dimerizes or    trimerizes, you might need to consider modifying the 
> construct or expression strategy (including moving away from bacteria if 
> necessary). I would suggest against trying to rescue an inherently unhappy 
> construct or trying to make a protein in a system that does not fold it.
> 
> The high MW peak for the "free" MBP is a little strange. I produced MBP-TEV 
> cleavage site(ENLYFQG) fusion as my control before, never observed any 
> non-monomer species of it. the precision site does not seem any worse than 
> the TEV site by sequence. (But did you put a stop codon right after the 
> precision site? What is the exact sequence of your C-term tail? Also double 
> check your plasmid sequence for frame-shift mutations, since you have 
> modified the vector.) Forming heterogeneous aggregates of 10+ the monomer 
> size is indicative of misfolding if it is not caused by a hydrophobic tail. 
> For robust proteins such as MBP, such bad aggregation suggests that something 
> in your expression or purification procedure needs to be optimized. Inducing 
> for too long (eg, >3hr at 37C) or harsh lysis can contribute to misfolding.
> 
> One more question: does your protein contain cys residues? Are they supposed 
> to be oxidized or reduced?
> 
> Zhijie
> 
> 
> 
> -----Original Message----- From: Xiao Xiao
> Sent: Wednesday, November 13, 2013 5:20 PM
> To: [email protected]
> 
> Subject: [ccp4bb] Oligomerization of maltose-binding protein
> 
> Dear all,
> 
> I am purifying a fusion protein with maltose-binding protein(MBP) as the 
> N-term tag. As a control, I also purified free-MBP from empty vector. The 
> vector I used is pMAL-c5x (from NEB) with an addition of precision protease 
> cutting site (8 residues, LEVLFQGP) between MBP and multi-cloning site.
> 
> Then I run both protein through Superose6(size-exclusion chromatography) to 
> check oligomerization states. To my surprise, both protein has two peaks, one 
> is at the monomer size and another one has a big MW. SDS-gel shows from both 
> protein, the small peaks are both free-MBP monomer, but the big peaks are 
> fusion protein and free-MBP respectively. The estimated MW of the big peaks 
> are similar to 10 folds of their corresponding monomers but heterogeneous 
> (checked by DLS and native gel).
> 
> To my knowledge, MBP should behave as a monomer. Does anyone have seen the 
> similar thing before or could give some explanations? I suspect maybe the 
> precision protease cutting site and the following multi-clonging site at the 
> C-term of free-MBP might cause oligomerization, could it be possible? I am 
> trying to delete this part to see whether it will improve, is there any other 
> thing I can try in order to eliminate heterogeneity?
> 
> Any suggestion or comment will be really appreciated.
> 
> Thank you!
> 
> Best regards,
> 
> Xiao Xiao 
>  

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