Cindy said:
<After the vet checked her over, he said that in his opinion one of the
tendons around the mitro valve had ruptured.  This would explain the sudden
collapse and the weakness she was having.

She is almost 14 years old (Dec) and has been diagnosed last Oct with a
grade 3/4 murmur.  My vet says the murmur is the same.>

Cindy,

I am very sorry this happened to Sabrina.  I know how special she must be to
you at almost 14 years old.  My Savannah collapsed one evening, and I rushed
her to the ER thinking it was a ruptured chorda tendineae.  The vet did not
think so.  After much testing in the weeks to come, it turned out to be
cancer.  I'm not suggesting that Sabrina has cancer, just that it might not
necessarily be her heart.  I hope Sabrina is doing well this evening.  (I
love the name Sabrina.  I have a cat with that name.)

Re: Ruptured Chorda Tendineae:  In the textbook, "Small Animal Cardiology
Secrets," by Jonathan A. Abbott, DVM, he says:

"What catastrophes can explain sudden clinical deterioration in patients
with MVD?

It should be recognized that CHF in veterinary patients often has an
apparently sudden onset.  Many pet dogs are relatively sedentary; because of
this, cardiac disease can progress unnoticed until a piont when even minimal
exertion results in severe dyspnea.  Additionally, subtle changes in
respiratory rate and character can be difficult for owners to recognize.
These and possibly other factors delay recognition of CHF in animals until
it is well advanced.  However, patients with MVD are subject to catastrophes
that can result in acute decompensation.

Rupture of a chorda tendineae is a relatively common acute complication of
MVD; the severity of the resultant clinical signs is dependent on the
functional importance of the ruptured chord and the compliance of the left
atrium. Rupture of a first-order mitral chorda tendineae causes acute and
severe mitral valve regurgitation and the resultant increase in left atrial
pressure may result in fulminant pulmonary edema that is refractory to
medical therapy.  If a third-order chord ruptures in a patient with a
compliant and capacious atrium, it may go undetected.  In fact, ruptured
chordae are sometimes found on postmortem examination of patients with MVD
that succumb to extracardiac disease.  Rupture of mitral chordae is most
common in patients that have preexisting mitral valve regurgitation and
associated cardiac enlargement.  The result is clinical decompensation, the
severity of which is determined by factors stated above.  Occasionally,
rupture of a first-order mitral valve chorda is observed in patients that
have only mild mitral valve disease.  In these cases, the acute elevation in
left atrial pressure is catastrophic and severe pulmonary edema results.
This is one of the few clinical scenarios in veterinary medicine that
results in truly acute heart failure; radiographically, the cardiac
silhouette is only minimally enlarged in the presence of florid edema.
Sometimes the pulmonary hypertension that develops subsequent to acute
increases in left atrial pressure can result in right-sided CHF manifest
clinically as ascites.

Rupture of the left atrium is an uncommon complication of MVD.  Although
surgical treatment of atrial rupture has been described, most often the
result is death due to tamponade.  Rarely, rupture of the atrial septum
results in an acquired atrial septal defect."

Carol Richards
East Tennessee, USA
[EMAIL PROTECTED]
http://www.geocities.com/carolscavaliers/
"My little dog, a heartbeat at my feet."
                      Edith Wharton

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