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To determine whether JAK/STAT pathway is involved in proliferation of
hypereosinophilic syndrome (HES) cells, and reveal the pathogenesis of HES;
observe the dynamic change of the clinical phenotype and hematological response,
the _expression_ of janaus kinase/signal transducer and activator of transcription
(JAK/STAT) protein or FIP1L1-PDGFRA mRNA in one HES patient treated with
low-dose imatinib.
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