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Anticancer agents that interfere with microtubulin function are in
widespread use in man and have a broad spectrum of activity against both
hematological malignancies and solid tumors. The mechanisms of actions of these
agents have been better defined during the past decade, indicating that there
are distinct binding sites for these agents and that they interfere with
microtubulin dynamics (growth and shortening of tubules) at low concentrations
and only evoke microtubulin aggregation or dissociation at high
concentrations.
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