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The Poxviridae family members vaccinia and variola virus enter mammalian
cells, replicate outside the nucleus and produce virions that travel to the cell
surface along microtubules, fuse with the plasma membrane and egress from
infected cells toward apposing cells on actin-filled membranous protrusions. We
show that cell-associated enveloped virions (CEV) use Abl- and Src-family
tyrosine kinases for actin motility, and that these kinases act in a redundant
fashion, perhaps permitting motility in a greater range of cell types.
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