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Scientists have uncovered an unexpected
functional link between a phosphatase known to act as a tumor suppressor and
BCR/ABL, a kinase that is intimately linked with cancer development and
progression in specific forms of leukemia. The study, published in the November
issue of Cancer Cell, provides fresh insight into the molecular mechanisms that
are involved in induction and progression of chronic myelogenous leukemia (CML),
and suggests a new strategy that may be an effective therapy for both
treatment-sensitive and treatment-resistant forms of CML.
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