-Caveat Lector-
Neurological Impact Of
Fluoride Toxicity
>From Gayle
[EMAIL PROTECTED]
6-13-1
While the links from fluoride consumption to cancer,
osteoporosis, and other physical ailments are long
established, recently studies have shown that fluoride
impact is much broader and includes neurological and
cerebrovascular effects.
Fluoride has been banned in Sweden, Norway, Denmark,
West Germany (now unified), Italy, Belgium, Austria,
France, and The Netherlands.
Tea (not including the herbal varieties) is a surprising
culprit, in addition to fluoridated drinking water supplies
and fluoride toothpaste.
A 52-week study of the factors that enhance or inhibit the
bioavailability of aluminum and its effects on the nervous
system was published in 1998 in the Journal of Brain
Research. According to the report, the equivalent of
fluoridated drinking water in terms of elemental fluorine
levels had an impact on brain tissue similar to the
pathological changes found in humans with Alzheimer's
and other forms of dementia.
The introduction to the report noted, "One of their most
remarkable findings was that animals administered the
lowest dose of aluminum-fluoride (0.5 ppm) exhibited a
greater susceptibility to illness and a higher incidence of
mortality than the animals administered the higher levels
(5 ppm, 50 ppm) of aluminum [without the fluoride].
"While the small amount of aluminum-fluoride in the
drinking water of rats required for neurotoxic effects is
surprising, perhaps even more surprising are the
neurological results of the sodium-fluoride at the dose
given in the present study (2.1 ppm) [the amount used to
achieve 1 ppm of elemental fluorine used in fluoridation].
"In most reports of chronic fluoride toxicity, the data
provided are usually limited to weight loss, dental and
skeletal changes, indicators of carcinogenesis, and
damage to soft tissues.
"Fluoride has diverse actions on a variety of cellular and
physiological functions, including the inhibition of a
variety
of enzymes, a corrosive action in acid mediums,
hypocalcemia [low blood calcium], hyperkalemia [excess
blood potassium], and possibly cerebral impairment."
The authors summarize, "Chronic administration of
aluminum-fluoride and sodium-fluoride in the drinking
water of rats resulted in distinct morphological alterations
of the brain, including the effects on neurons and
cerebrovasculature."
A previous study by Mullenix, et al. in Neurotoxicology and
Teratology, 1995, documents abnormal behavioral
responses by animals exposed to fluoride at various
stages of gestation, which resulted in permanent
hyperactivity if exposed prenatally, or extreme lethargy if
exposed after birth, with some animals not able to find
their way out of a circular maze to the same food source
every day.
This study of the neurological effects of sodium fluoride,
which is commonly touted as a safe and even
health-promoting drinking water additive, came on the
heels of a recent report in the Journal of the American
Medical Association that 2 million people a year become
ill, and more than 100,000 die, from medicines judged by
the medical community to have been "correctly prescribed
and correctly administered."
The fluoride/aluminum association is of particular
importance as it relates to Alzheimer's Disease. Aluminum
by itself is not readily absorbed by the body. However,
fluoride ions combine with aluminum to form aluminum
fluoride, which is absorbed by the body. In the body, the
aluminum eventually combines with oxygen to form
aluminum oxide or alumina. Protein bound to alumina in
afflicted brains forms the plaques and tangles
characteristic of Alzheimer's disease.
In a study by Dr. Robert Isaacson at the State University
of New York, aluminum fluoride was added to rats' diet.
This, contrary to normal expectations, passed through the
brain barrier and gave the rats short-term memory loss,
smell sensory loss, unsteady gait, and loss of structures
of the neo-cortex and hippocampus, all symptoms of
Alzheimer's. A Varner and Jensen study conducted with
Isaacson confirmed this in 1998.
B-12 AND FOLATE CAN REDUCE ALZHEIMERS
RISK<#TABLEtop Elderly people with low blood levels of
vitamin B-12 and folic acid (folate) may face an increased
risk of developing Alzheimer's disease. Vitamin B-12 plays
an important role in maintaining nerve cells, and some
research has linked low blood levels of the vitamin to
Alzheimer's and mental decline. Few studies have looked
at whether there is such a connection between
Alzheimer's and folate, a B vitamin key to the production
and maintenance of body cells.
The May 8, 2001, issue of Neurology reports on a study
of 370 individuals aged 75 and older, in which
investigators found that those with low levels of either
vitamin were twice as susceptible to Alzheimer's over a
3-year period as those with normal levels. The link was
even stronger among study participants who performed
well on mental tests at the start of the study.
The reason for the link is unclear, but low blood levels of
B-12 and folate can lead to elevations in the amino acid
homocysteine, which may in turn damage nerve cells, the
authors note. Vitamin B-12 is found in meat, fish, eggs,
and milk. Vegetarians are advised to supplement their
B-12 intake. Folate occurs naturally in leafy green
vegetables, dried beans and peas, and citrus fruits,
among others. Many cereals are fortified with folic acid,
the synthetic form of folate.
Alzheimer's is the most common form of dementia,
affecting an estimated 4 million Americans. The exact
cause remains elusive, but scientists believe genetics and
environmental factors conspire to trigger the onset of the
disease.
The May 8, 2001 issue of Neurology Magazine listed the
following as warning signs of Alzheimer's Disease:
memory loss that affects job skills; language problems;
difficulty performing familiar tasks; misplacing objects;
changes in mood and behavior; poor judgment;
disorientation as to time and place; personality changes;
problems with abstract thinking; and loss of initiative.
Brain imaging is recommended to help rule out other
causes of memory loss or dementia. This includes
computed tomography (CT) and magnetic resonance
imaging (MRI). Genetic testing has not been found
particularly useful. [Annual Meeting of the American
Academy of Neurology, 2001]
The elderly are not the only ones that need to worry about
B-12. According to one practitioner, the most common
cause of B-12 deficiency is a vegetarian diet.
The drug Prilosec (omeprazole) has been shown to
decrease B-12 absorption [Annual of Pharmacotherapy,
May 1999]. This is possibly due to its effects on
decreasing the production of intrinsic factor, which is
needed for proper B-12 absorption. Other medications
may have similar adverse effects.
There is also little question now that B-12 and folic acid
are useful to reduce homocysteine and the associated
increase in heart disease and birth defects.
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