December 7, 1999 

Neurotoxic agents used in Seattle gas attacks? 

In addition to eye witness accounts that some Seattle 
"police" were armed with M-16s, some doctors are concerned 
that tear gas and pepper spray were not the only chemical 
agents used against demonstrators. 

Perhaps the Special Forces "advisors" CNN reported being 
present in Seattle a more hands on approach than we 
know. 

    Neurotoxic Symptoms In Chemical Warfare Casualties At 
Seattle WTO Meeting        by kirk james murphy, MD 
     <[EMAIL PROTECTED]> 10:04am Tue Dec 7 '99 

        Direct Action Network Medical Collective members 
provided first aid and other  care for thousands of 
casualties of chemical warfare during the Seattle WTO 
protests. Collective members identified a subset of 
casualties with  symptoms consistent  with exposure to 
neurotoxins which disrupt  acetylcholine irritant 
agents do not explain the symptoms. 

              The Direct Action Network Medical 
Collective was organized to provide first aid and 
other health services to activists and others 
(onlookers, media, residents, and anyone else 
requesting assistance) in Seattle for the WTO  meeting 
last week. The Collective members  provided care for 
literally hundreds of chemical warfare casualties on 
both  November 30 and December 1; hundreds more have 
requested information and treatment in subsequent 
days. As one may imagine, Collective members have 
acquired  extensive expertise in the recognition  and 
treatment of symptoms arising from exposure to pepper 
spray (OC) and "tear gases" (CS and CN). 

            Individuals exposed to chemical weapons in 
the late afternoon and evening of           December 
1st at two locations downtown  blocks adjacent to Pike 
Place Market and   the Seattle neighborhood of Capitol 
Hill  evinced and reported a pattern of  symptoms 
which is inconsistent with the  pattern of symptoms 
which may be ascribed  to irritating agents. This 
"atypical"  pattern of symptoms includes the rapid 
onset of: 

mydriasis (pupillary dilation) with 
resultant impairment of visual acuity; 

            tachycardia (rapid heart rate) with some 
palpitations; 

           new-onset hypertension (high blood 
pressure) in one individual; 

           nausea, vomiting, diarrhea (persisting  for 
days after exposure); 

           abrupt or immediate onset of menstruation 
(asynchronous with usual menstrual 
          cycle); 

          muscular fasciculation (twitches); 

          muscular dyscoordination; 

         lethargy, confusion, disorientation, 
diminished concentration, nocturnal 
         hallucinations. 
         Moreover, some casualties reported an  abrupt 
experience of loss of muscular   tone and strength 
that sometimes (but not always) immediately preceded a 
loss of  consciousness; one observer of these 
affected individuals reported uncontrolled, spasmodic 
movements in  those affected. 

        Some individuals exposed in the Pike Place Market 
area reported that the  aforementioned symptoms came 
immediately  after exposure to a non-irritating agent 
which was did not cause pain, lacrimation, or burning 
on mucous  membranes. 

        At least one individual complaining of 
fasciculations and cognitive impairment   days after 
exposure reported resolution of both symptoms for a 
duration of   approximately five to six hours within 
twenty to thirty minutes after treatment with 10 mg of 
the specific antidote dicyclomine (Bentyl) - an 
anticholinergic  medication which crosses the 
blood-brain  barrier and hence can relieve central 
nervous symptoms of excessive cholinergic activity as 
well as relieving peripheral  symptoms of the same 
etiology. 

          The pattern of symptoms is not consistent 
with known mechanisms of action of the irritant 
chemical weapons OC, CS, or CN. The pattern, however, 
is consistent with  disruption of neurotransmitter 
activity. Lamentably, the single most compelling 
explanation for the observed findings is  the 
(deliberate or accidental) inclusion   of 
"incapacitating agents" which disrupt  neuronal 
function in the chemical  munitions discharged by law 
enforcement  agencies in Seattle during the WTO 
protest. 

         While direct cholinergic effects or indirect 
(inhibition of acetylcholinesterase) effects arising 
from synergistic combinations (of OC, CS, and CN) 
cannot be ruled out at this time,  the experience and 
observations  Medical Collective members, together 
with  the aforementioned information, appears  to most 
robustly support the hypothesis   that the casualties 
described above  resulted from exposure to 
cholinesterase      inhibitors used as chemical 
weapons in crowd control. 

        The Seattle Post-Intelligencer reported on December 4 
that the Seattle Police   Department had to replenish 
its chemical weapons stocks by going to outside 
sources. Various individuals have  reported being told 
by individual law enforcement officers that chemical 
weapons in addition to OC, CS, and CN   were deployed 
by various entities; these  anecdotal accounts are not 
yet confirmed. 

        Any information regarding the use of  chemical 
munitions in addition to OC, CS  or CN, as well as 
information regarding  the discharge of chemical 
weapons by   agencies other than the Seattle Police 
Department would be helpful. 

            Available Internet information from 
presumably authoritative (eg, DOD)   sources describes 
"nerve gases" as lethal  agents. While the casualties 
described   above - fortunately - have not included 
fatalities, the apparent pattern of  excessive 
cholinergic activity and the   fact that 
"incapacitating agents" is the  term of art for a 
subset of chemical weapons used in crowd control 
suggests  that the term "nerve gas" may be a 
suboptimal descriptive term.  "Incapacitating agents" 
or "neurotoxic   agents" may be more precise ways of 
describing chemical munitions with the  effects 
described above. 

        If you were exposed to chemical weapons  during the 
WTO protests and have the  pattern of "atypical" 
symptoms discussed  below, please make a written, 
signed, and dated account of your exposure, including 
details such as the (approximate)  location in which 
you were exposed and  the date and part of the day 
(morning,    midday, afternoon, evening) of your 
exposure, as well as the nature of your     symptoms. 
Please send such accounts to  the email above and to 
the ACLU unit    investigating law enforcement actions 
in Seattle during the WTO protests. 

        Collection of discharged chemical weapons  munitions 
is proceeding apace at this time. Collection of serum 
samples from affected individuals will probably be 
organized over the next 48 hours. Assay  of garments 
(and other materials) exposed    to the chemical 
warfare agents on  individuals demonstrating the 
aforementioned pattern of symptoms is also 
contemplated. If you have canisters  or other chemical 
munitions containers  discharged during the WTO 
protest, please  contact the above email address for 
information about how to forward these  containers for 
chemical analysis 
[NOTE: Canisters of "CS" or "CN" (tear gas) or   "OC" 
(Oleoresin Capsaiscum) are not   needed - we know they 
were used. The   containers to be sent for chemical 
analysis would only be those without the  above 
markings or those with markings  consistent with the 
use of other agents.] 

         Your comments are invited and desired.  Obviously, 
the escalation of chemical  weapons used in "crowd 
control" to include even non-lethal neurotoxins is an 
exceedingly grave prospect: the qualitative leap from 
irritant agents  (OC, CS, CN) to agents which disrupt 
neuronal function is truly an appalling  prospect. 
This communication is intended  to ensure the fullest 
possible discussion  and consideration of the events 
and  mechanisms described above. 
 

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