Hi Chris,

You can probably find all that you need here

It looks like it is a defunct research programme, but maybe you could
follow up citations.

I could probably dig out an e-copy of the ECAL paper from my
institution's Springerlink subscription, if you're really interested.

Further comments interspersed

On Wed, Aug 07, 2013 at 01:03:36AM +0000, chris peck wrote:
> Hi Prof. Standish
> Unfortunately my subscription to Athens ran out a long time ago and I don't 
> have access to the paper you mention.
> I'm still not sure you've addressed the crux of the argument. Lets say you 
> have a bunch of codons that when processed by a replicating mechanism spit 
> out a bunch of amino acids. Lets say the replicating system isn't optimized 
> and has low redundancy so that
> codonA -> aa1
> codonB -> aa2
> codonC -> aa3
> Now there is a random mutation in the mechanism that ought to offer some 
> redundancy:
> codonA -> aa1
> codonB -> aa1
> codonC -> aa2
> codonD -> aa3
> Unless there has been a concomitant mutation in the DNA strands the
mechanism will process, this 'optimization' is in fact catastrophic. 

That is what I was referring to as the boundary being unstable. The
two schema cannot coexist at the same location. What I had in mind was
that they existed contemporaneously, but in different physical
locations - eg different rock pools perhaps.

ISTM that you are implictly assuming that these replicating
hypercycles only emerged once, whereas I would think that replicating
RNA probably arose many times quite easily when life wasn't around to
gobble them up.


Prof Russell Standish                  Phone 0425 253119 (mobile)
Principal, High Performance Coders
Visiting Professor of Mathematics      hpco...@hpcoders.com.au
University of New South Wales          http://www.hpcoders.com.au

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