Suppressed T cell-mediated immunity in patients with COVID-19: A clinical retrospective study in Wuhan, China https://www.journalofinfection.com/article/S0163-4453(20)30223-1/abstract
Does anyone here have a sense for the types of conditions (cf covid19 comorbidities) that cluster around all 3 proteins? My naive googling returns comments about graft-vs-host and various autoimmune conditions and immunosuppressive therapies. But I'm having trouble getting a feel for how they might all cluster together. Maybe it's simply that lower amounts of *any* of the 3 subsets produces a higher risk for those with covid19? But I keep hearing that the "cytokine storm" is more than just an effect, it may loop back and be part of the problem. -- ☣ uǝlƃ .-. .- -. -.. --- -- -..-. -.. --- - ... -..-. .- -. -.. -..-. -.. .- ... .... . ... FRIAM Applied Complexity Group listserv Zoom Fridays 9:30a-12p Mtn GMT-6 bit.ly/virtualfriam unsubscribe http://redfish.com/mailman/listinfo/friam_redfish.com archives: http://friam.471366.n2.nabble.com/ FRIAM-COMIC http://friam-comic.blogspot.com/
