Hi Bob Larson, Hi,
Liver and the kidneys both are involved in the excretion of silver. I suggest http://www.silver-colloids.com/Papers/AltmanStudy.PDF on Silver excretion from the body. Silver Dust ranges from very large particles to microscopic. In Scandinavia, Silver Reclaimation workers have been observed in 20 year plus studies. They were in perfect health. The organ damage cited was from a study of Mine workers who beside being exposed to Silver were also exposed to Cadmium (known heavy metal poison injurious to kidneys). The Toxicological Registry Report notes this and says that silver and kidney damage in this case has to be discounted. The writers that cite this 1990 study for kidney damage are committing the sin of omission, or at least are taking part of the quote out of context to support their position. You can find the study at the CDC website if you know where to look. It is peer reviewed. Colloidal Silver taken orally causing injury: Mirsattari SM, et al. Myoclonic status epilepticus following repeated oral ingestion of colloidal silver. Neurology 2004;62:1408-10. Researchers concluded that the neurotoxicity and the resulting death were due to ingestion of colloidal silver. This one case and only of Silver and Seizures (status epilepticus, continuing seizures) dealt with a 71 year old man who took Colloidal Silver and FOUR VITAMIN SUPPPLEMENTS over a 4 month period. How much CS, what PPM, and what type of CS was never addressed in the study. The 4 neurologists had the man on plasmapheresis as his blood levels in tests showed high silver levels. After being treated (to remove the silver) the man still in a coma, and died 5 1/2 months later. Status epilepticus is defined as recurrent or continuous seizure activity lasting longer than 30 minutes in which the patient does not regain baseline mental status, in other words regaining consciousness. In the United States, the overall mortality is 10-15%. In people who are note epilectic, its first appearance normally indicates a tumour, abscess, or traumatic brain injury. The after the first 5 minutes of the seizure, the damage can be sufficient to prevent the patient from reawakening on their own. The 5 doctors writing the above paper published in Neurology concluded it was the silver that induced the status epilepticus. Here is what the doctors wrote. Mind you they did not mention the supplements the man was taking as a possible contributory factor: "The authors report a case of a 71-year-old man who developed myoclonic status epilepticus and coma after daily ingestion of colloidal silver for 4 months resulting in high levels of silver in plasma, erythrocytes, and CSF. Despite plasmapheresis, he remained in a persistent vegetative state until his death 5.5 months later. Silver products can cause irreversible neurologic toxicity associated with poor outcome." Argyric Rats have been studied for years as to silver accumlation in the brain, silver excretion, etc. Concerning these lab rats, one would think that seizures would have been observed from high concentrations of silver given to these animals at all stages of life and a relationship between silver and seizures would have been established. My questions, What were the additional supplements the man was taking? If none invasive methods (MRI) were used did the MDs look for a Tumor? (I am assuming that neither injury, abcess, or tumors were found, considering the number of MDs on the case. Although Doctors have been known to overlook the obvious.)What other Rx medications was the senior on?Was the man taking pilocarpine for Glaucoma? This drug has been used to induce SE in laboratory rats? (Pilocarpine, or Pilocar usage is limited by its toxic side effects or adverse reactions. The drug has been suggested to test the effects of Germ warfare damage in animals. Did he use Aspartame? N-methyl-d-aspartate is known to induce SE in rats.Was a post mortem was performed? (In Calif. those under a doctor's care at time of death do not require a PM.)As emergency personal had been called, was CPR used. Generalized myoclonus (a type of SE) is commonly seen in comatose patients following CPR and is seen by some as an indication of catastrophic damage to the neocortex.Was the cause of death from SE or the drugs used to treat SE? "On February 20, 2008, a fourteen year old girl who had been seeing a neurologist, but had not been diagnosed with epilepsy had a convulsive seizure at her school that lasted less than five minutes, and an ambulance was called because nobody present was able to properly handle the situation. She was transported to the hospital, but after an abnormal amount of time she still had not awakened. After EEG electrodes were attached to her head, it was discovered that she was suffering from status epilepticus. Her doctor went through the routine normally used to stop an episode of status epilepticus, but the seizure would not stop. In the end she had to be given such a large amount of anti-convulsive medications that her breathing system shut down and she went into kidney failure. After extreme measures (Ventilators, dialysis), she survived and recovered, returning to school in 10 days." A long quote from wikipedia slightly shortened. I hope this answers some of your question
