Good post Steve. dee On 29 Jan 2010, at 22:56, Norton, Steve wrote:
> > In analyzing Dave's data and reviewing again the Altman study I believe > that I have found (or more accurately, stumbled upon) the reason why EIS > is safer to use than other forms of silver. It also explains why EIS is > both processed the same as other silver compounds, primarily by the > liver, and also differently at the same time. I will go into that > shortly. I think you will find it interesting. Either as a long sought > after answer or as an opportunity to tell me what an idiot I am. > > But first, a request. I think that some good information can be gained > from evaluating the silver usage of those on the list who have blue > moons in their fingernails. I would like to request all who can, to > provide it the following info: > > Average daily amount used. > Average ppm of EIS > How EIS was made > Number of years till nail beds turned blue/grey > Any unusual supplements used in conjunction with the EIS > > The more data we have the better we can know how to best use EIS. While > I think that there are few things we can conclusively draw from Dave's > experience, one item think I feel is encouraging is that there appears > to be a significant difference between the time blue moons appear and > when argyria might occur. And maybe we can use Dave's timeline as a > rough predictor for someone with blue moons to develop more serious > signs with or without changes in usage. And yes, it would be very > speculative but it might be better that what we have now - nothing. > > For those who might wish to brush up on the Roger Altman Silver > Elimination Study, here is a link to Roger's report. > http://www.silver-colloids.com/Papers/AltmanStudy.PDF > > > And here is a discussion of the study and silver elimination in general: > http://www.info-archive.com/colsil%20silvertox.ht > > > First, I would like to discuss how much silver the liver can the liver > excrete in a day. When silver compounds in the blood exceed that limit > it leads to increased deposition of silver in the tissues. The excess > silver has to go somewhere. The following study puts that at about 1 mg > of silver per day (or 3.4 oz of 10 ppm CS): > > "In a study involving biologic monitoring of workers (n = 37) in one of > the silver smelting and refining industries in which the exposure is > entirely > by inhalation, silver was found in the blood (0.011 :g per milliliter > [mL]), > urine (<0.005 :g/mL), and feces (15 :g/g). Control subjects excreted > about > 1.5 :g/g in the feces (n = 35). The author suggests that human fecal > excretion > of silver at exposure levels equal to the Threshold Limit Value (TLV) > (0.1 mg per cubic meter [m3]) would be about 1 mg of silver per day (Di- > Vincenzo et al. 1985)." > > If you exceed this limit continuously over a period of time you will > have excess silver deposited in the tissues. For non EIS silver > compounds, it has been found that roughly 10% of the silver ingested is > absorbed into the bloodstream. That would indicate that you can take up > to 34 oz of 10 ppm CS per day and not saturate the biliary excretion > path. However, the Altman study shows that 100% of ingested EIS enters > the bloodstream and so EIS should be limited to 3.4 oz of 10 ppm EIS, or > equivalent, to remain within the bilary excretion capability of the > liver. However, should you exceed that amount, EIS is uniquely processed > by the body and that is where EIS becomes safer to use and less likely > to cause argyria. > > To understand what happens when you take more EIS than the liver bilary > excretion path can handle, you need to look at the Altman study. > > The Altman study starts by Roger Altman taking 2.34 mg of silver, in the > form of EIS, daily for an extended period of time. That amount is > significantly higher than the 1 mg daily amount the liver can process > and caused a buildup of silver in Roger's system. Roger then stopped > taking any EIS and then measured over a 96 day period the excretion of > silver out through the liver (feces) and the kidney (urine). Over the 96 > day period, the silver excreted through the feces was fairly constant > but varying around 1.5 mg per day. I believe that this represents the > excretion capacity of Roger's bilary excretion path and is consistent > with the study referenced previously. > > What is interesting, and what sets EIS apart from other forms of silver, > is what was happening in the kidney. For silver compounds other than > EIS, only a little silver is excreted through the urine. Even when there > is excess silver in bloodstream or when the bilary excretion path is > blocked and preventing the liver from excreting silver. But the Altman > study shows a large amount of silver excreting out in the urine. Usually > in the 3 to 4 mg per day range, but at times as much as 10 mg. It is > this result that has erroneously led people to believe that EIS is > primarily excreted through the urine. > > Out at the 96th day samples, the silver in the urine has dropped to 0.64 > mg but the excretion through the feces was 2 mg, indicating that the > bilary excretion path was still eliminating silver at its maximum > capacity while only a little silver was still passing out through the > kidney. > > I propose that the primary excretion path for EIS is through the liver > EXCEPT when the amount of silver from EIS in the blood exceeds the > liver's capacity to excrete it AND AS LONG AS THE SILVER IS IN SOLUTION. > Why do I say that? It is clear that EIS is somehow unique from other > types of silver. We can tell that by the fact that it is processed > differently as shown by the study. But what is that difference? I > propose that it is that EIS forms silver chloride in the stomach acid. > And that silver chloride has a low solubility in the blood. I also > propose that silver chloride does not have to go into solution to pass > into the bloodstream. But that the silver chloride molecule is small > enough to pass into the blood while still a particle. > > Small particles in the blood are filtered by the kidneys. When the > silver chloride exceeds the solubility capability of the blood, it > exists in the blood as particles and is filtered out by the kidney. It > is this unique excretion path that makes EIS less likely to cause > argyria. However, EIS is not impervious to causing argyria. As long as > silver content in the blood is higher than what the liver can process, > some of that silver is being deposited in the tissues. It is just that > EIS significantly reduces the problem. > > It also makes me think that the problems with using non distilled water > and additives to the water is not caused so much by the silver chloride > that is formed but by other silver compounds formed with other > impurities. > > - Steve N > > > > > -- > The Silver List is a moderated forum for discussing Colloidal Silver. > > Instructions for unsubscribing are posted at: http://silverlist.org > > To post, address your message to: [email protected] > > Address Off-Topic messages to: [email protected] > > The Silver List and Off Topic List archives are currently down... > > List maintainer: Mike Devour <[email protected]> > >
