Dear Mr. Rosser,
I offer the following in answer to your question:
Please understand my answers are from memory...from
studies done 3 and 4 years ago, but they should be reasonably accurate for
discussion purposes. Our investigators found that the further down the gut
CS progresses, the less the concentration---and effectivity. Except in one
case where a modified catheterization was performed for installing a
moderate-diameter tube in the gut mid-section of a dog (for access to
passing contents) no "instream" samples were taken from ANY higher mammals.
Rats were considered improper subjects because of their extremely high
metabolic rate and pronounced variability of moisture content of solid fecal
material.. The dog evaluations (from stomach to mid-gut) revealed an almost
linear decrease in surviving CS concentrations considered therapeutic for
acute afflictions.....as solid material reached the mid-gut regions. This
phenomenon was directly reflective of the moisture loss in the waste-train.
Only in cases of very high CS administration (10 ounces daily of 5 ppm per
40 lbs body weight), did therapeutic levels of silver survive the journey to
evacuation stages. Even then, due to a lack of homogeniztion of the fecal
field, plus the very low moisture fraction......total control of pathogenic
agents appeared unlikely. However, many pathogenic agents require as much
moisture as does CS......to be effective against their target organisms.
This said, there was a very pronounced difference between both the number of
different pathogens, their individual populations and vigor.....occurring
between control animals----and subject animals receiving CS (even in mild
concentrations). Therefore, the conclusion may be drawn that "some"
prophylactic response (in the lower gut) does accrue from the use of
CS.....employed over a moderate time period (two to three weeks in extent).
One interesting observation of possible value; CS (except as a
bacteriostatic) had no appreciable affect on the bulk of
pathogens.....UNLESS in an environment of at least 30% moisture content
(about like an ink-blotter surface 10 seconds after blotting a 1" diameter
(fresh) ink splotch).
As I have never committed the name of the "silver-loving"
bacteria to memory, you will have to wait until I can interrogate someone at
the laboratory, who has access to the data file. I will do this ASAP and
post the name. I do remember an interesting characteristic this bacteria
was supposed to have displayed----after several generations of isolation (
10, I believe) from silver contaminated soils, the bacteria lost its
immunity to the effects of silver.
I hope this information is of value to you.
Sincerely, Brooks Bradley.
russ e rosser wrote:
> BROOKS BRADLEY <[email protected]> writes:
>
> > quantities as low as 1.5 ounces were successful in a "flooding
> > coverage" of the
> > duodenum and upper stomach---in dogs---within 45
> > seconds
>
> > In fact, alimentary
> > insults are
> > the MOST EASILY controlled of any we have ever evaluated.
>
> Could you pls. comment on CS' ability to reach the *lower* GI tract at
> effective concentration levels, before diffusing into the bloodstream
> through the upper?
>
> > Excepting one (non-pathogenic) bacteria---which
> > proliferates
> > in silver solutions, we have never tested colloidal silver on a
> > pathogen.....which it failed to control.
>
> What is that pathogen, pls? I'd be interested to know (and so might the
> World Health Org., for *other* reasons) HOW it withstands CS. Tx.
>
> --Russ
>
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