` Dear Suzy,
We have, in the past, conducted quite
extensive experimental evaluations of both systemic--and topically
applied--- substances for addressing a wide variety of bacterial and/or
venous stasis insults....with variable results..By far, the most
effective composite protocol we instigated involved a combination of
protocols. In many cases of protracted insults, the challenge of
detritus tissue particles must be addressed. The most effective---and
pain-tolerant---procedure included the dilution of 5 to 10 ppm CS with
20% Lidocaine (2% strength) being applied to the "already dampened"
surface of the ulcer/sore. After---and ONLY after---5 to 8 minute
interval, the surface was physically debrided (if required by
circumstances). Note: Most surface wounds/sores/ulcers deep enough to
involve underlying tissue will have exposed a large field of nerve
sensors. Pain from almost any type of physical contact is, usually,
INTENSE. Therefore, debridement always included some form of pain
abatement.
Following debridement, fluids weeping from
the surface was carefully blotted and the chosen protocol instituted.
Our most effective one included a preliminary flushing with 3.5% H202.
The actual method included the use of a simple "squeeze-bulb" or
ear-syringe to apply the H202. The liquid was gently applied in copious
quantities and allowed to bubble for about 15 seconds; then blotted
with gauze and repeated 4 or 5 times---or until the "foaming action"
decreased to trace levels. Using a portion of the CS X Lidocaine
solution, we diluted with Full-strength DMSO at the rate of 10% DMSO to
90% CS X Lidocaine solution....by volume. This is the material used as
the resident dressing. The insult was thoroughly dampened with this
solution and the entire area enclosed in a suitable fashion (we used
high-quality, clear garbage-type bags)
and pure O2 applied via tubing (at low inflating pressure...usually 12
to 15 psi). Additional 02 was only applied as the bag showed evidence
of collapsing from pressure loss. i.e. Just restoration of acceptable
inflation is all that was required. The 02 atmosphere was maintained
for from 30 to 45 minutes (usually). After removing the 02 bag, a
six-fold gauze bandage (thorought soaked in the CS X DMSO X LIDOCANE
solution) was applied to the affected area. This bandage was large
enough to extend at least .75" beyond the edges of the insult.One little
amendment which proved to be quite helpful involved the cutting out of a
small clear-plastic patch which followed the general outline of the
edges of the actual ulcer. This patch was carefully placed "directly"
over the ulcerated area (but on the OUTSIDE of the gauze bandage). The
plastic "hydrator" and the gauze bandage were held in place using
high-quality adhesive-tape, employing either and X pattern or a top and
bottom edge restraint pattern. It is important to use tape long enough
to attach to dry areas considerably beyond the edges of the bandage.
This will facilitate holding the bandage in proper physical alignment.
There were several factors involved in
determining the frequency in which the bandages were changed---and or
02 support employed. Among SERIOUS/ACUTE volunteer presentations, one
of two circumstances generally prevailed: (1) the subject was exposed
to the entire protocol every four hours (during the daylight period) and
left with the last bandage in place until the following morning;. (2)
the subject was exposed to the H202 protocol and the 02 topical
hyperbaric-type treatment, only, on schedule of every 3 hours, with at
least one hour intervals between treatments; but no treatments between
10:00 pm and 8;30 am.
Generally, after 2 or 3 days, only the (1)
protocol was utilized----and then in a modified manner. To wit: after
completion of the complete protocol, the gauze bandage was left in
place---without disturbance---for three days (however, fresh CS X
Lidocaine X DMSO solution was applied every 2 to 3 hours during the
daylight hours. The bandage WAS NOT disturbed during thses
activities. As healing progressed, the time-table was modified to even
longer intervals.
There is some other, ancillary, information of
consequence.....but I must go now. I will try to follow-up sometime
tomorrow.
This protocol, and/or slight variations, proved
successful in a consequential number of cases which had shown ABSOLUTELY
NO measureable improvement from a multitude of other differing
protocols.
Sincerely, Brooks
Bradley.
[email protected] wrote:
> This is the 4th or 5th case of flesh eating bacteria in this area in a
> short
> time. Would CS, the hyperbaric system described by Brooks, or
> anything else
> we have talked about, have worked for this guy? He is the dad of a
> friend.
> suzy
>
> http://www.dmregister.com/news/stories/c4788996/14655842.html
>
> Flesh-eating disease attacks Urbandale man
>
>
>> A sudden attack of "flesh-eating disease" leads to amputation.
>
> By KATE KOMPAS
> Register Staff Writer
> 05/11/2001Joseph Rogers of Urbandale got out of bed about 6 a.m. on
> April 21
> and couldn't feel his toes.Doctors removed his leg before noon.Rogers,
> 76,
> fell victim to necrotizing fasciitis, commonly known as the
> "flesh-eating
> disease."The disease is caused by a variation of the bacteria that
> causes
> strep throat and is as rare as it is deadly. The bacteria typically
> enter the
> body through a wound and immediately begin to devour muscle and fat.
> It can
> progress at a speed of three centimeters an hour, doctors say.The
> Centers for
> Disease Control reports there are fewer than 2,000 people infected in
> the
> United States each year. About 100 of them will die. Experts say the
> chance
> of contracting necrotizing fasciitis from another person is near
> nonexistent."I've got a disease I can't pronounce the name of," Rogers
> said
> Thursday. "I don't understand why this happened to me."His was the
> second
> case of necrotizing fasciitis reported in Iowa in the past month.
> Curtis
> Benttine, a truck driver from St. Ansgar, was hospitalized earlier
> this week.
> Twelve pounds of infected tissue was removed from Benttine's leg.Dr.
> Cort
> Lohff of the state health department said statewide numbers on
> necrotizing
> fasciitis aren't kept, but the disease has shown up before:* Randy
> Schabaker,
> 51, of Des Moines died of the flesh-eating disease in March 1993.*
> Bryan
> Crawford, 12, of Independence lost part of his leg to the disease in
> 1994.*
> Ryan Johnson, 14, of Independence died in 1995. He also had leukemia.*
> Stuart
> Eliasen, a 34-year-old Plainfield farmer, died in 1997.* Matthew
> Potter, 6,
> of Amana died in 1998 from a streptococcus infection that later was
> determined not to be the "flesh-eating" form.When his toes went numb,
> Rogers
> called to his wife, Mary, who thought he was having a stroke and
> called 911.A
> bruise appeared on his left leg by the time he arrived at the
> hospital,
> Rogers said. The discoloration spread, inching up his leg within a few
> hours.
> By 10 a.m., the doctors at Iowa Methodist Medical Center said they
> should
> amputate.Doctors still are puzzled over how Rogers contracted the
> disease."I
> cried for three days," Mary Rogers said Thursday. "But I've accepted
> it."Mary, who's been married to Joseph for 53 years, has been reading
> everything she can on the disease.The retired steel worker and World
> War II
> veteran will start rehabilitation soon and will be fitted with a
> prosthetic
> leg."There's no pain," he said. "The good leg feels good, and the bad
> leg's
> not there." Rogers said. "I guess I'm lucky I got through it."
>
>
