As some members may be aware, polyvalent inhibitors
have been the subject of rather intensive investigation during the past
12-13 months. The objective being attempts to design anthrax antitoxins
and/or vaccine-like compounds. In reviewing an article which appeared
in "The Scientist" (16[12]:34, June10, 2002, I was intrigued to observe
a comment near the end of the article which stated "the C2 toxin
secreted by clostridium botulinum, the bacterium which causes botulism,
also assembles on the host cell surface and could potentially be blocked
by a dominant negative-mutant, as could the VacA toxin secreted by
heliobacter pylori, the bacterium that causes gastric ulcers."
Instantly, it occurred to me that since our investigations involving
Colloidal Silver controlling in vitro (and some in vivo cases involving
accidental contact with botulinum) colonies of clostridium botulinum had
been so successful.......by simple extension, the prospect for CS
proving equally effective for Anthracis----looks brighter still.
This article is the first....among any of the more
prestigious periodicals, to offer information allowing a direct
extrapolation for protocols affecting pathogens kin to Anthracis to be
considered. At least it is the first I have encountered.
Although somewhat "gun-shy" about posting urls (in view
of my horrendous editing errors of the past), I will attempt to post,
correctly, the link to "The Scientist" article.
Http://www.the-scientist.com/yr2002/jun/research2_020610.html
Hopefully, it will come through.
Sincerely, Brooks Bradley.
p.s. We enjoyed outstanding control of in vivo heliobacter pylori
(using CS) in 98% of all volunteers......during our ulcer studies some
three years ago.
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