Very interesting. Then any silver chloride ends up becoming silver albumin (like egg whites, or in fact MSP!), in the blood stream and silver metal. The precipitation of it onto particles is not a surprise given the ph and natural developers that are in blood, and if there were not a bunch of "seeds" available for this in the EIS already, could lead to argyria if silver chloride is taken alone.
Marshall Matthew McCann PE wrote: > In 1939 Hill and Pillsbury published their exhaustivereview of the > literature on argyria and the pharmacologyof silver. On page 13 of > their book, they discussedthe work of Minz(1930) on these issues > concerning the formof silver as it is it is absorbed and transported > in thebody. [Minz, B.: Ztschr. f. Klin. Med. 114:623-41, 1930] Hill > and Pillsbury wrote: "...Gager and Ellison state that the chemical > changeswhich silver undergoes previous to absorption are notclear, and > that the initial form in which silver isadministered is of no > importance as regards the finalcompound which will be transported by > the bloodstream.It has been assumed that a free silver ion entering > thebloodstream forms a soluble colloid with the plasmaprotein. The > compound in which silver is absorbed...hasbeen variously regarded to > be the albuminate, chlorideor reduced metal. The best available > experimental work on this subject hasbeen done by Minz who injected > Collargol into thebloodstream of a rabbit and analyzed the blood > atintervals for silver salt, silver albumin, and metallic silver.He > found that after one hour the plasma showed adecrease in silver salts > and an increase in the silverpresent in combination with albumin. The > amount ofsilver present as the colloidal salt diminished much > morerapidly than did the silver albumin and he concluded thatupon > injection into the bloodstream the silver waschanged to the albuminate > quite rapidly, and taken upby the organs in this form." Matthew
