Prouve ? Ce serait sympa que l'on prouve l'efficacit� d'un m�dicament assez anodin.

Pour la premi�re �tude, le r�sum� ne donne pas les m�thodes. C'est une petite s�rie et 
je me demande si on peut faire une �tude "d'�quivalence" avec un si petit effectif, 
avec une puissance suffisante ? Les auteurs ne disent pas dans le r�sum� la rapidit� 
et intensit� du ralzentissement. Et quelle dose comment ? Tu as les d�tails ?

Pour le second, ils disent "cliniquement peu significatif. Donc �a ralenti mais pas 
beaucoup. Tu as les d�tails ?


>Messsage du 08/01/2002 14:17
>De :  <[EMAIL PROTECTED]>
>A :  <[EMAIL PROTECTED]>
>Copie � :
>Objet : URG-L: Le magn�sium est efficace pour ralentir une FA
>
> Le magn�sium est efficace pour ralentir une FA (par action de frein sur
> le noeud AV). Ces deux r�f�rences le prouvent. Certains d'entre vous
> l'utilisent d�j� ?
>
> 1: Chiladakis JA, Stathopoulos C, Davlouros P, Manolis AS. Intravenous
> magnesium sulfate versus diltiazem in paroxysmal atrial fibrillation.
> Int J Cardiol 2001 Jul;79(2-3):287-91
>
> BACKGROUND: Drugs currently available for the acute treatment of
> paroxysmal atrial fibrillation have significant limitations. We assessed
> the safety and effectiveness of intravenous magnesium sulfate versus
> diltiazem therapy in patients with prolonged episodes of paroxysmal
> atrial fibrillation. METHODS: In a prospective randomized trial, 46
> symptomatic patients presenting with paroxysmal atrial fibrillation were
> given intravenous magnesium sulfate (n=23) or diltiazem (n=23) therapy.
> Primary outcome measures were effects on ventricular rate control and
> proportion of patients restored to sinus rhythm at 6 h after initiation
> of treatment. RESULTS: There were no differences in baseline
> characteristics between the two groups. Both forms of treatment were
> well tolerated, with no adverse clinical events. Both drugs had similar
> efficacy in reducing the ventricular rate at the first hour of treatment
> (P<0.05) with a tendency toward a further decrease during infusion times
> of 2 (P<0.01), 3, 4, 5 and 6 h, respectively (P<0.001). However, at the
> end of the 6-h treatment period, restoration of sinus rhythm was
> observed in a significantly higher proportion of patients in the
> magnesium group compared with the diltiazem group [13 of 23 patients,
> (57%), versus five of 23 patients, (22%), P=0.03]. CONCLUSIONS:
> Magnesium sulfate favorably affects rate control and seems to promote
> the conversion of long lasting episodes of paroxysmal atrial
> fibrillation to sinus rhythm, representing a safe, reliable and
> cost-effective alternative treatment strategy to diltiazem.
>
> 2: Eur J Emerg Med 2000 Dec;7(4):287-90 Magnesium efficacy in magnesium
> deficient and nondeficient patients with rapid ventricular response
> atrial fibrillation.  Eray O, Akca S, Pekdemir M, Eray E, Cete Y, Oktay
> C.
> We assessed the effect of magnesium sulphate (MgSO4) on lowering the
> rate in ventricular atrial fibrillation (AF), and evaluated the effect
> of this therapy in magnesium (Mg) deficient and nondeficient patients.
> This experimental clinical study was performed on 34 patients with rapid
> AF (ventricular rate [VR] > 120/minute) presenting to the emergency
> department of a tertiary care university hospital. Patients with
> systolic blood pressure < or = 100 mmHg, Hb level < or = 11.8, saO2 of <
> or = 96%, BUN > or = 40 or creatine > or = 1.8 were excluded (n = 15).
> Nineteen patients were given an initial 2 g MgSO4 bolus i.v. and a 1
> g/hour continuous infusion over 6 hours. To evaluate the presence of Mg
> deficiency, urine was collected from the onset of treatment and
> continued for the next 24 hours, and the excretion rate of administered
> Mg was calculated. Ventricular rates were obtained at baseline, after
> MgSO4 bolus, and every 15 minutes for the first hour. The decrease in
> the VR was statistically significant at 15, 30 and 60 minutes after Mg
> therapy (p = 0.0025, p < 0.001, p > 0.001). There was no difference in
> the response to Mg therapy between Mg deficient and nondeficient
> patients at 15, 30 or 60 minutes after therapy (p = 0.41, p = 0.28, p =
> 0.08). It is concluded that i.v. MgSO4 has a statistically significant
> but clinically limited effect on VR and this effect did not differ
> between patients with and without Mg deficiency.
>
>

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