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Scientists Connect Alzheimer's Disease To Mercury
International Academy of Oral Medicine and Toxicology
An Organization Dedicated to Evidence Based Health Care
P.O. Box 608531, Orlando, FL 32860-8531
T: 407-298-2450 * F: 407-298-3075 * Email: [EMAIL PROTECTED] *
Internet: www.iaomt.org
NEW RESEARCH CONNECTS MERCURY TO ALZHEIMER’S DISEASE!
Research conducted at the University of Calgary Faculty
of Medicine has demonstrated that trace amounts of
mercury can cause the type of damage to nerves that is
characteristic of the damage found in Alzheimer’s
Disease. The level of mercury exposure is consistent
with those levels found in humans with mercury/silver
amalgam dental fillings. The exposure to mercury caused
the formation of “neurofibrillar tangles,” which are one
of the two diagnostic markers for Alzheimer’s Disease.
The scientists found that other metals, including
aluminum, did not cause the damage. Previous research
has shown that mercury can cause the formation of the
other Alzheimer’s Disease diagnostic marker, “amyloid plaques.”
The research, published in a peer-reviewed medical
journal, is accompanied by a video visual presentation of the
effect. Utilizing digital time-lapse photography,
this video shows rapid damage to the nerve cells after
introduction of minute amounts of mercury. Funding
for this video was provided by the International Academy
of Oral Medicine and Toxicology (IAOMT).
SPECIAL NOTE: The University of Calgary has placed a copy
of the video on their site at:
http://movies.commons.ucalgary.ca/mercury/
To view the video you will need "Quick Time" Player which
can be dowloaded from the site if you do not have it.
This video will be available to media contacts (only) through:
Miss Karen Thomas
Media Relations
University of Calgary, Faculty of Medicine
T: 403-220-2945 F: 403-210-8141 Email: [EMAIL PROTECTED]
Media Embargo Date: 26 March 2001
Title: “Retrograde Degeneration of Neurite Membrane
Structural Integrity of Nerve Growth Cones Following In
Vitro Exposure To mercury.”
Authors: Leong, CW; Syed, NI; Lorscheider, FL.
Journal: NeuroReport, 12(4):733-737, 2001.
BIOPROBE COMMENT: This study should remove all doubt
regarding the role that dental mercury from
amalgam fillings plays in the development of Alzheimer’s
Disease (AD). Although the American Dental
Association would like to have you believe otherwise,
science has clearly demonstrated that there is a positive
correlation between brain mercury levels and the number
and surfaces of “mercury/silver” amalgam dental
fillings. The mercury levels that caused the devastating
damage to nerve cells in the above referenced study were
100 to 1000 times below those found in the brains of people
with “mercury/silver” amalgam dental fillings.
In 1997, researchers at the University of Calgary Medical
School and the College of Pharmacy at the University of
Kentucky clearly demonstrated that exposing rats to the
same levels of mercury vapor that can be released from
“mercury/silver” amalgam dental fillings caused the
mercury to interact with brain tubulin and disassemble
microtubles that maintain neurite structure. The identical
neurochemical lesion of similar or greater magnitude is
evident in Alzheimer brain homogenates from approximately
80% of patients, when compared to human
age-matched neurological controls.
(Neurotoxicology 1997;18(2):315-324)
In 2000, researchers at the Neurobiology Laboratory,
Psychiatric University Hospital in Basel, Switzerland using
neuroblastoma cells exposed to mercury demonstrated
an increase in production of amyloid protein that makes
up the amyloid plaques as well as significantly increasing
the phophorylation of Tau protein. (J Neurochem 2000
Jan;74(1):231- 236)
Studies demonstrating a correlation between amalgam
dental fillings and brain mercury levels:
Lakartidningen 1986 Feb 12;83(7):519-522
Swedish Dental Journal 1987;11(5):179-187
Sci Total Environ 1987 Oct;66:263-268
J Prosthet Dent 1987 Dec:58(6):704-707
FASEB J 1989 Dec;3(14):2651-2646
Sci Total Environ 1990 Dec 1;99(1-2):1-22
Sci Total Environ 1993 Sep 30;138(1-3):101-115
J Trace Elem Med Biol 1995 Jul;9(2):82-87
Zentralbl Hyg U:mweltmed 1996 Feb;198(3):275-291
FASEB J 1998 Aug;12(11):971-980
Biometals 1999 Sep;12(3):227-231
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