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Fanconi anaemia (FA) is a genetic disease characterised by bone marrow
failure with excess risk of myelogenous leukaemia and solid tumours. A widely
accepted notion in FA research invokes a deficiency of response to DNA damage as
the fundamental basis of the 'crosslinker sensitivity' observed in this
disorder. However, such an isolated defect cannot readily account for the full
cellular and clinical phenotype, which includes a number of other abnormalities,
such as malformations, endocrinopathies, and typical skin spots.
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