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Chronic myeloid leukemia (CML) is characterized by the presence of a
t(9;22)(q34;q11.2), which leads to the well-known BCR-ABL1 fusion protein. We
describe a patient who was diagnosed clinically with a typical CML but on
cytogenetic analysis was found to have a t(9;22)(p24;q11.2). Chromosomal
fluorescence in situ hybridization showed that the BCR gene locus spanned the
breakpoint at band 22q11.2 but that the ABL1 gene was not rearranged.
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