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Stem cell shift may lead to infections, leukemia
Aging of blood-producing stem cells could be responsible for the relatively
high incidence of infections and myeloid leukemia in the elderly.



Stem cell shift may lead to infections, leukemia
Christen Brownlee

Researchers have long wondered why elderly people suffer more infections and
have a greater chance of developing myeloid leukemia, a type of blood
cancer, than younger people do. Now, research in mice suggests that the
aging of blood-producing stem cells could be responsible for both
conditions.

With age, the body of a person or other animal loses its capacity to sustain
its tissues and organs. "Since we know the cells mediating this maintenance
are stem cells, it doesn't take a great leap of faith to think that stem
cells are at the heart of that failure," says Derrick Rossi of Stanford
University.

To examine whether the aging of stem cells contributes to infections and
leukemia, Rossi and his colleagues irradiated young and old mice to kill off
their blood-making stem cells. The scientists then transplanted such stem
cells from young donor mice into elderly irradiated animals and from old
donors into young irradiated animals.

After several weeks, the researchers found that young animals' stem cells
transplanted into the old mice produced the different types of blood cells
in ratios much like those in young mice that haven't been irradiated.
However, the young animals that received old animals' stem cells had
significantly fewer new lymphoid blood cells-which make cells that battle
infections-than normal young animals do.

After examining gene activity in the stem cells transplanted from old
animals, Rossi's team found a boost in activity among genes responsible for
creating myeloid cells. These create red blood cells and some other blood
components. Many myeloid-production genes have been associated with myeloid
leukemia in people.

The scientists conclude in the June 28 Proceedings of the National Academy
of Sciences that a shift from lymphoid-cell production to myeloid-cell
production could be responsible for the increases both in infections and in
risk of leukemia that come with old age.

References:

Rossi, D.J., et al. 2005. Cell intrinsic alterations underlie hematopoietic
stem cell aging. Proceedings of the National Academy of Sciences 102(June
28):9194-9199. Available at
http://www.pnas.org/cgi/content/full/102/26/9194.

Further Readings:

Schlessinger, D., and G. Van Zant. 2001. Does functional depletion of stem
cells drive aging? Mechanisms of Ageing and Development 122(Sept.
30):1537-1553. Abstract available at
http://dx.doi.org/10.1016/S0047-6374(01)00299-8.

Sources:

Derrick J. Rossi
Department of Pathology
Stanford University School of Medicine
279 Campus Drive
Stanford, CA 94305


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