Re: DHEAS

Sat, 13 Dec 2003 19:38:49 -0800

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DHEAS

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From: MinnieChat

Labcorp explains it as follows:
 
</FIELD-LIST><FIELD-LIST name="Use"> </FIELD-LIST><FIELD-LIST name="Methodology"> </FIELD-LIST><FIELD-LIST name="Additional Information">
Reference Interval</FIELD-NAME>
<FIELD-TEXT>See table.1,2
Age  Male
(�g/dL) 		 Female
(�g/dL) 
1-7 d  91-376  73-367 
8-15 d  37-224  44-247 
1-3 y  <30  <79 
4-6 y  <186  <38 
7-8 y  <94  <68 
9-10 y  <75  <160 
11 y  <152  <98 
12 y  <344  <177 
13 y  <242  <167 
14 y  <286  32-301 
15 y  59-310  39-288 
16 y  47-357  58-354 
17 y  102-341  97-399 
18-19 y  108-441  145-395 
19-29 y  280-640  65-380 
30-39 y  120-520  45-270 
40-49 y  95-530  32-240 
50-59 y  70-310  26-200 
60-69 y  42-290  13-130 
>69 y  28-175  17-90 
Tanner Stage 
I  <87  <65 
II & III  <151  <175 
IV  75-282  57-230 
V  121-368  76-378 
</FIELD-TEXT>
<FIELD-NAME>Use</FIELD-NAME>
<FIELD-TEXT>Work up women with infertility, amenorrhea, or hirsutism, to identify the source of excessive androgen; aid in the evaluation of androgen excess (hirsutism and/or virilization), including Stein-Leventhal syndrome and adrenocortical diseases, including congenital adrenal hyperplasia and adrenal tumor. DHEA-S is not increased with hypopituitarism. It is low in Addison disease.</FIELD-TEXT>
<FIELD-NAME>Methodology</FIELD-NAME>
<FIELD-TEXT>Immunochemiluminometric assay (ICMA)</FIELD-TEXT>
<FIELD-NAME>Additional Information</FIELD-NAME>
<FIELD-TEXT>DHEA sulfate is the major steroid of the fetal adrenal. DHEA-S is the principal adrenal androgen and is secreted together with cortisol under the control of ACTH and prolactin. DHEA-S is elevated with hyperprolactinemia.

Elevated levels may be found in the adrenogenital syndrome3 or adrenocortical neoplasms or hyperplasias. In females and children, DHEA excess causes masculinization.

Increased 3--androstanediol glucuronide indicates excessive androgen in peripheral tissues. Persistent anovulation, the polycystic ovary or Stein-Leventhal syndrome is characterized by increases of circulating levels of testosterone, androstenedione, dehydroepiandrosterone and DHEA-S. 17-hydroxyprogesterone and DHEA-S are only mildly increased compared to cases of adrenal hyperplasia. Patients with androgen-producing adrenal tumors also have moderate increases of 17-KS.

Testosterone is derived from ovaries, adrenals, and the peripheral tissues. Increased DHEA with normal testosterone provides evidence for an adrenal cause of excessive androgen. Low levels are found in amniotic fluid in Down syndrome.4</FIELD-TEXT>

Hope this helps - Minnie

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