Hi Roger,

I am not entirely clear what happens to CS in the digestive tract, I
have trouble finding reliable information...more work to be done. I am
still of the view that only Ag ions can pass into the blood stream.


The solubility constant of AgCl (Ksp) = 1.8 x 10^-10
When concentration [Ag+] = [Cl-] then [Ag] dissolved = 1.34 x 10^-5 M

Ksp = [Ag+][Cl-]

10.8ppm CS contains 1.0 x 10^-5 M so that is at the limit of solubility
with an equivalent amount of Cl- .

Higher concentrations of Cl- (product) gives higher concentration of the
reactant (AgCl), known as the common ion effect.

The concentration of [Cl-] in HCl at pH2 is 1 x 10^-2 M
[Ag-] = 1.8 x 10^-10 / 1.0 x 10^-2 = 1.8 x 10^-8 M

So only 1/500th to 1/1000th of our initial 10ppm CS will remain
dissolved, the rest is passed on as insoluble AgCl, although higher
temperatures increase the solubility and I am as yet unsure on the
effect of the H+ ions and other enzymes, proteins and so on. Also if the
concentration of Cl- anions drops in the duodenum the solubility of the
Ag+ ions will increase.

What do you make of it?

Ivan.

----- Original Message -----
From: <[email protected]>
To: <[email protected]>
Sent: Saturday, 25 November 2000 09:48
Subject: CS>Production of Silver Chloride at pH 2


> Ivan: You said, "I do believe that particles of metalic silver will be
> ionised in the
> acid stomach environment, and that clumps of ions will be broken
apart,
> but I doubt that at low pH that silver will combine with Cl- to form
the
> silver cloride molecule, much in the same way that silver nitrate in
> solution is not combined in molecules but exists as free silver ions
and
> free nitrate ions."
>
> Ivan: Isn't the formation of AgCl molecule or solid at pH 2
(presumably from
> ionized clumps of CS) based on the solubility product of AgCl at pH 2?
Do you
> have data that suggests that under the anticipated concentrations of
Ag+ and
> Cl- that the AgCl(s) solubility product will not be reached? Roger
>
>
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