Caspi's work on depression, published in Science (which I believe is peer 
reviewed :-)) a few years back, seems to be a good example. He showed that 
people with a short allele of a specific gene linked to serotonin production 
was related to depression, but only when people experienced significant 
negative life events. I'll try to dig up the reference.

________________________________________
From: [EMAIL PROTECTED] [EMAIL PROTECTED]
Sent: Tuesday, March 25, 2008 10:03 AM
To: Teaching in the Psychological Sciences (TIPS)
Subject: Re:[tips] The Gene Illusion

I said:

>> >I wonder if Mike could favour us with a high-quality research study
or
> >two published in a peer-reviewed journal which provides evidence of such
> >gene-environment interactions. Surely his belief must be based on
> >evidence.

Mike Palij obligingly replied:

> The classic study that answers Stephens request is the discovery of
> phenylketonuria or PKU, the genetic defect that prevents the metabolism
> of phenylalanine and leads to development of mental retardation.

Ah, yes, excellent example. How could I have forgotten that one, which is
indeed a classic. But it is a specialized choice and far from our current
discussion of social and genetic factors in the development of children
into adults. The defective gene impairs a biochemical pathway.
Consequently phenylalanine, which is an abundant component of everyone's
diet, becomes harmful. It is extraordinarily difficult to provide a diet
which is free of this substance, and only a severely abnormal and
artificial one will do. Kids hate it so much that it becomes a major
challenge to keep them on it.

By analogy, I would concede that a social environment as abnormal as a
severely-phenylalanine-low diet is could have lasting effects, and you
wouldn't even have to specify a particular genotype. But to do it, you
might have to raise the kid with wolves.

How about a particular genotype and a particular type of environment
which kids might be exposed to which is not artificially constructed and
imposed,  as the low-phenylalanine diet is. If we have to reach back to
the PKU diet developed in the 1950's for an example, what does this tell
us about how many more relevant gene-social environment examples have
been identified since then?

Stephen

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Stephen L. Black, Ph.D.
Professor of Psychology, Emeritus
Bishop's University      e-mail:  [EMAIL PROTECTED]
2600 College St.
Sherbrooke QC  J1M 1Z7
Canada

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