http://www.bipolarodyssey.com/infections.htm Thorazine inhibits candida 
species. (Yeast infections are implicated in autism, leaky gut, psychosis.) 
Calmodulin activity has been identified in retroviruses. This is 
particularly important because as stated in the book, retroviruses are now 
implicated in schizophrenia.   Like Lyme disease, Toxoplasma gondii is more 
prevalent in psychiatric patients than the general population. Further, 
drugs used in the treatment of schizophrenia and bipolar disorder inhibit 
the replication of Toxoplasma gondii.  We tested 12 neuroleptic compounds 
and found that of these, the antipsychotic haloperidol and the mood 
stabilizer valproic acid most effectively inhibit Toxoplasma growth in 
vitro. Several medications used to treat schizophrenia and bipolar disorder 
have the ability to inhibit the in vitro replication of T. 
gondii."Intravenous calcium replacement can cause mania; calcium 
supplementation can increase mania; calcium channels blockers reduce mania. 
An errant enzyme linked to bipolar disorder, in the brain's prefrontal 
cortex, impairs cognition under stress, an animal study shows. The disturbed 
thinking, impaired judgment, impulsivity, and distractibility seen in mania, 
a destructive phase of bipolar disorder, may be traceable to over activity 
of protein kinase C (PKC)   ...
The above suggests that it may be necessary to conceptualize bipolar 
disorder not as a monolithic "psychiatric"  disorder, but as a collection of 
heterogeneous biological disorders that can be individually identified and 
treated. Infection-mediated-excessive PKC may be one such disorder. ...  The 
above description does not do justice to the complexity of processes 
involved.  It describes only one process involving a biomarker of mania, 
excessive PKC.   While PKC is a major player, there is no single explanation 
for the mechanisms of mania. In fact, there are many up and down regulated 
proteins in the brains of those who have bipolar disorder, suggesting that 
no single explanation is sufficient to account for manic symptoms.

http://www.mombu.com/medicine/heart/t-diseases-of-the-mind-bipolar-disorder-1466983.html
For its part, the parasite Toxoplasma gondii, which can be found in 
undercooked meat and cat feces, can lead to full-blown psychotic episodes.
Some studies suggest that the parasite stimulates the production of a
chemical similar to LSD, producing hallucinations and psychosis. Even when
the parasite lies dormant in muscle and brain tissue, it can affect
attention span and reaction time in otherwise healthy people.  Few
children avoid coming down with a streptococcus infection, also known as
strep. Scientists now think that one in 1,000 strep sufferers also develops
abrupt-onset obsessive-compulsive disorder (OCD) in a matter of weeks. Strep
bacteria trigger OCD by igniting an overzealous response from the immune
system, which attacks certain types of brain cells, causing inflammation.
Symptoms generally die down after a few months but can flare up again,
especially if theres another bout of strep, says Susan Swedo, a
childhood-disease expert at the National Institutes of Health. The most
effective treatment, still experimental, is to filter out the misbehaving
antibodies from the blood. Best is to treat strep early on.
The specter of a depression germ or contagious obsessive-compulsive
disorder is unnerving, but it also opens up many more treatment
options antibiotics, vaccines, checking for ticks. Geneticists believe that
diseases may trigger the onset of inherited mental illnesses by activating
key genes. Avoiding and treating infection may be just as important as the
genes you inherit, and a whole lot easier to do something about.


http://www.genhealth.com/Products/vitaklenz/parasite_the_silent_killer.htm

Microscopic parasites can get into your joints and eat the calcium linings 
of the bone. This can lead to excruciating arthritis
Parasites can eat the protein coating on the nerves (the myelin sheath). 
This causes a disruption in the nerve signal from the brain.
Many times when a person has an inflamed appendix, it is removed and found 
to be loaded with parasites!
Story for you, DB: Story #4: One woman was working in her garden. She had 
recently taken a special herbal soup to rid her body of parasites. She was 
weeding the plants when she noticed dozens of barely visible red parasites 
on her arm. She at first thought they came from the plants. They didn't. 
They were coming out of the pores of her arms.



How can I repair damage to the nerve sheath>  ... If you get reinfected with 
parasites they will usually attack the same place they attacked before. . . 
. Crossed eyes are almost always caused by parasites in the eye focus 
muscles. . . .    Parasite damage to the heart most often afects either the 
aortic valve or the mitral valve.  Damage to the heart valves causes skipped 
beats, irregular beats, rapid beats, angina, shortness of breath, chest 
pain, arm pain, upper back pain and neck pain.   . . . Parasite activity 
often causes adhesions to form in veins.    Vein valves are often destroyed 
or damaged by parasites causing an extra load on the heart.
All vaccines developed with the use of bovine serum are suspect for being 
contaminated with Nanobacaterium. Dr Overman found them in vens, heart, 
muscles, sinuses, bones, joints, adrenals, kidneys, brain, nerves, lungs, 
ears, eyes and spleen.  He believes they can attack any tissue.
Strongyloides roundworms can be a nightmare. ... eggs can hatch in 30 
minutes after coming in contact with warm moist skin. .. Constant 
reinfection is the rule. In the skin they tunnel through the tissues causing 
intense burning, pain and itching.  One client had over eight thousand in 
his skin.  People may also develop allergies to the worms, which increases 
inflammation, swelling and itching.   Sometimes large worms such as ascaris 
adults living in sinus cavities must be removed manually by an ear, eye, 
nose and throat specialist. ...  Parasitic worms often invade the brain of 
the fetus  in utero causing retardation.  ... Killing parasites once does 
not stop you from getting reinfected. .. the new parasites invariably go 
right back to the same organ that was infected previously.
 
http://books.google.com.au/books?id=Yk7KCPshHIIC&pg=PA245&lpg=PA245&dq=nerve+sheath+parasite&source=web&ots=gotKD4Ezia&sig=yjx-kj0oRDA7hcT_0QGS-fHxVS4&hl=en&sa=X&oi=book_result&resnum=10&ct=resulthttp://www.drovermannd.com/index.cfm?CFID=436819&CFTOKEN=70960484In
 diabetes, worms are in the pancreas. With epilepsy, they are in thebrain. In 
multiple sclerosis, they are what is eating the myelin sheaths offthe exposed 
nerve endings. (It is a misconception that the immune system ofthe body is 
attacking itself in 
MS)http://healing.about.com/cs/uc_directory/a/uc_parasites_2.htmIn my practice, 
I always find parasites and worms associated with thefollowing diseases: 
Diabetes, hypoglycemia, juvenile diabetes, anorexia,asthma, depression, anemia, 
chronic fatigue syndrome, AIDS, cancer, tumors,anorexia, bulimia, colitis, 
epilepsy, hyper-activity syndromes, learningdisabilities, baldness, prostate 
trouble, appendicitis (the appendix is afavorite place for parasites), elevated 
white blood counts, Hodgkin'sdisease, Multiple Sclerosis, psoriasis, some 
excemas, leukemia, lymphoma,arthritis, leprosy, ulcerative colitis, low 
hemoglobin, poor blood quality,iron deficiencies and most bowel disorders.For 
example, cancer is simply a symptom indicating a body that isauto-intoxicated 
with parasites, worms, yeast, virus and other bacteria, andwhose colon lacks 
beneficial flora.http://eands.caltech.edu/articles/LXVI4/brainworms.htmlIn 
fact, millions of people worldwide are infected by these efficaciousbrain 
parasites. Many of these brain parasites cause debilitating conditionsand 
sometimes even death.Two parasites with disease-causing capabilities arethe 
pork tapeworm, Taenia solium, and the amoeba Naegleria fowleri. Inaddition to 
their medical importance, these two organisms illustrate themany ways that 
brain parasites are able to affect their hosts through theirmethods of invasion 
and survival. ... But in order to reach the brain fromthe bloodstream, the 
larvae must traverse the blood-brain barrier.Unfortunately, researchers s!
 till don't know exactly how this happens. Manyscientists think that the larvae 
can release enzymes that are able todissolve a small portion of the blood-brain 
barrier to allow the parasite toget through into the brain. ... If the larvae 
attach to the brain tissue,then the host often experiences seizures. This 
occurs partly because thepresence of the larvae causes the activity of the 
brain to become wild anduncontrolled, thereby causing a seizure. On the other 
hand, if the larvaeattach to the brain-fluid cavities, the host experiences 
headaches, nausea,dizziness, and altered mental states in addition to seizures. 
Theseadditional symptoms occur because the flow of the brain fluid is blocked 
bythe larvae. Often, the presence of the larvae also causes the lining of 
thebrain-fluid cavities to become inflamed, further constricting the flow ofthe 
brain fluid. Since the cavities are a closed system, blockage of thecavities 
exerts pressure on the brain. This increased cranial pressureforces the heart 
to pump harder in order to deliver blood to the brain area,increasing the 
pressure on the brain even more. If the condition is nottreated, the heart 
eventually cannot pump enough blood to the brain, neuronsbegin to die off, and 
major brain damage occurs. ... For instance, theparasites are able to prevent 
the complement proteins from attaching totheir surfaces. The tapeworms can even 
release molecules that act as decoys,tricking the killer proteins into leaving 
them alone. The cysts also releaseother proteins that are able to protect them 
from being eaten, although howexactly this is accomplished is still unknown. 
There is some evidence thatthese proteins are able to prevent phagocytes from 
accurately targeting thecysts. One of the ways that phagocytes are able to go 
to the right place inthe body during an infection is by following a chemical 
trail. This trail isproduced by other immune cells at the site of infection. 
Some of theproteins released by the cysts are able to obscure this chemical 
trail sothat the phagocytes be
 e lost on their way to the infection. Cysts arealso thought to release a 
second set of proteins that decreases the activityof new phagocytes. These 
proteins affect another group of immune cells thatcontrol the activity of new 
phagocytes; these regulatory immune cells thendecrease the number of active 
phagocytes. Finally, a third set of proteinsreleased by the cysts is thought to 
be able to prevent phagocytes fromproducing the proteins necessary to kill the 
cysts. Tapeworms, however, diewhen the host does, and so they try very hard to 
keep from being detected bythe immune system. And in fact, they do a fairly 
good job at that, sincemost tapeworm infections aren't noticeable until many 
years after thetapeworms get into the brain. The immune system is only able to 
have a bigeffect on the infection when the tapeworms start to die, often from 
oldage.... For instance, there is evidence that both parasites use enzymes 
topenetrate the blood-brain barrier, and both use a decoy strategy to 
deflectthe attention of the immune system.http://www.damninteresting.com/?p=399 
     A small minority of people havestrong psychological effects from 
toxoplasmosis, including delusions,hallucinations, and paranoia. Infected men 
appeared more jealous andsuspicious. Infected women appeared more warm-hearted 
and outgoingToxoplasma is associated with damage to the brain's astrocytes - 
glial cellsthat function as an interface between neuronal and non-neuronal 
tissues.Astrocyte damage has also been associated with schizophrenia.  Now add 
inthat pregnant women with high levels of Toxoplasma antibodies are morelikely 
to have children who later develop schizophrenia, and you havesomething to give 
most people pause. Torrey's study also found that some ofthe drugs used to 
treat schizophrenia have an inhibiting effect onToxoplasma growth.What exactly 
the connection is between Toxoplasma and schizophrenia has notbeen determined, 
though it seems clear there is one. What alterationsToxoplasma makes to human 
psychology in general is even more unclea
 Whatis abundantly clear is that whatever those alterations are, they affect 
ahuge number of people. Can we afford to be as ignorant of them as wecurrently 
are?On www.flickr.com/photos/jimfischer/182605629/   is what looks like a 
brainoverflowing with worms, but a poster said she found it was a 
dolphinstomach.  don't know.http://www.foxnews.com/story/0,2933,455067,00.html  
story of a womanoperated on for a tumour, but Drs found a worm in her brain 
instead. Has avideo, supposedlyNeurocysticercosis is acquired through 
consumption of food contaminated withfeces of a T. solium tapeworm carrier 
(i.e., through fecal-oral contract)(4,5,12). The life cycle of T. solium is 
shown in Fig. 1. Eggs of thetapeworm are shed in stool and contaminate food 
through poor hygiene. Whenthese eggs are ingested and exposed to gastric acid 
in the human stomach,they lose their protective capsule and turn into larval 
cysts, calledoncospheres. Oncospheres cross the gastrointestinal tract and 
migrate viathe vascular system to the brain, muscle, eyes, and other 
structures. Oncein the brain, the larval cysts (cysticerci) initially generate 
a minimalimmune response and may remain in the brain as viable cysts for 
years.http://www.pubmedcentral.nih.gov/articlerender.fcgi?art....Are there any 
links to the "egg sack" data?For really astonishing data about the pathogens 
and heavy metals thatmake it through the blood/brain barrier, check out the 
work of Y.Omura, M.D., a bona fide genius whose work is far, far too little 
known.> This is all very good, but the link between mental illness and brain> 
damage is too real to pass up. And that egg sacks have been found in> the 
brains of bipolar people is also instructive.>


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