When I consider why the same SSRI can work for different disorders
with different symptoms (to whatever extent it works better than
placebo), I try to bear in mind that the therapeutic effect isn't a
direct result of the acute SSRI action.  Rather, it seems to be a
result of the complex ways in which the brain responds to the SSRI
during the first few weeks of administration--a long list of
compensatory up- and downregulations of receptors.

And I take that to mean that the acute SSRI action is just a
well-placed kick on the side of a malfunctioning TV set.  It induces a
somewhat mysterious chain of events, and perhaps those events are
different in an anxious brain than in a depressive brain.

If you ever need to remind yourself that the acute actions of SSRIs
are relatively unimportant, consider the drug tianeptine.  It's an
effectve antidepressant/anxiolytic, but it's an SSRE--a selective
serotonin-reuptake ENHANCER.  Seems there's more than one way to kick
the malfunctioning TV set.

--David Epstein
  [EMAIL PROTECTED]

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