I would add that those who insist on modelling the atoms that they can’t see 
display a quasi-religious fervour about it, as if there is only one right way. 
Sorry about the snark.

One example I remember from my graduate student days is this:
https://tinyurl.com/bddefd3t

where intact and truncated gamma-deta resolvase crystallised in *exactly* the 
same crystal form, with two entire DNA-binding domains waving in the breeze as 
far as anyone could tell from dissolving the crystals.  For the 
build-all-the-sidechains approach to be intellectually consistent, as Esko 
pointed out you must do that for all disordered loops and domains, and the N- 
and C-termini unless you have cast-iron evidence (MS on the crystals) that 
these atoms are indeed not present.  If you are working on a glycosylated 
protein, you need to model at the very least all of the first attached NAG and 
NAM and (for preference) run detailed MS on the crystal(s) to identify which 
sites have what levels of glycosylations and model out to the minimal 
identified conserved core. Complete intellectual consistency would require a 
detailed analysis of the fractional extensions at each glycosylation site and 
partial occupancies for those atoms.

At least in the academic circles I have worked in, even some modellers will 
insist on starting from a PDB model with high ADP sidechains as if there is an 
implied truth to them - and often (again my experience, YMMV) turn to you with 
a great cry of ah-ha! that the MD says that they are wrong, so what use is 
crystallography anyway?  Their complete absence sends a rather clear signal to 
the people using the model that the people solving the structure haven’t a clue 
as to where those atoms are.

Operationally, our job is to come up with an accurate model for the atoms that 
we can see. At least that’s my belief. Everything else we can leave to the 
hallucinations (I use the word advisedly) of AlphaFold2 and its offspring.  So 
let’s live and let live?  You put yours in with high ADPs: I take them out.

In both cases, the (protein structural) world is not going to come to an end.  
We can rely on the Voldemort of modern diplomacy for that.

Adrian

On 17 Mar 2023, at 11:50, Esko Oksanen 
<[email protected]<mailto:[email protected]>>
 wrote:

Dear Manfred,

In addition to personal preference I think there are some philosophical 
differences in what the model actually means. I would argue that what we see in 
the density is stronger evidence of the chemical identity than what we believe 
we have used as a starting material. After all we don’t know what reactions can 
happen during crystallisation or even data collection. I would also argue that 
the model should not primarily represent the chemical composition as such, but 
rather those atoms that are actually ordered. So if there is no electron 
density, it is not evidence of that particular atom being absent, but it that 
doesn’t mean we should model it. If one would apply the same logic to hydrogen 
atoms we should include them in the model since there is no evidence of their 
absence. Similarly we could explicitly model water molecules in the disordered 
solvent and just let them refine to high ADPs, since we have no evidence of the 
absence of water molecules. Or any disordered region of the protein such as 
termini. By not modelling these termini, hydrogens or water molecules we could 
also give the impression that they are not there, but I think the users of the 
models can and should be expected to understand the limitations of the model. I 
would find it more misleading to model things that we don’t see but assume to 
be there versus not modelling them. I think if the “end users" of the model are 
educated enough that they notice unphysically high ADPs they will also 
understand a truncated lysine side chain.

  Just my 2c,
  Esko

On 17 Mar 2023, at 09:57, Manfred S. Weiss 
<[email protected]<mailto:[email protected]>> 
wrote:


Dear all,

many views have been expressed in this thread, which I have been
following with great interest. Unfortunately, I have to say that many
of the views are more based on personal preferences, than on the
scientific evidence behind.

Here are some facts, that one may want to consider:

1. When you have crystallized your protein, and collected data
from it, the obvious assumption is that it is still intact. Unless you
have additional information, e.g. from mass spec, that some
side chains are cleaved off, they are there.

2. Absence of evidence (meaning missing electron density)
is NOT evidence of absence !!!!

3. If you trim, you give the impression that the respective atoms
are not there. This means that the solvent model will be incorrect,
because the area will be defined as solvent.

In my view, the best approach is to build the side chains in their
most plausible conformation, or maybe in 2 or 3 or 5 different
conformations, and let the ADPs refine freely.

All the best
Manfred




<http://www.icm.uu.se/structural-biology/griese-lab/>























































































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